Saturday, May 15, 2010

Rabbit Hole: Part 12 Humanity Discovered

You may ask what the heck am i talking about.

This is very important about the rhesus factor and ethereal/plasma energy we all possess.

In the secret orders and societies they separate BLACKS AND WHITES??? WHY????

This is your stepping stone to understanding the RHESUS FACTOR???

TO BE CONTINUED GOD WILLING.

biology is very important listen up after you have read this i am about to explain in a recording of what this all means.

http://en.wikipedia.org/wiki/Adrenal_gland

Medulla

The adrenal medulla is the core of the adrenal gland, and is surrounded by the adrenal cortex. The chromaffin cells of the medulla, named for their characteristic brown staining with chromic acid salts, are the body's main source of the circulating catecholamines adrenaline (epinephrine) and noradrenaline (norepinephrine).

Derived from the amino acid tyrosine, these water-soluble hormones are major hormones underlying the fight-or-flight response.

http://en.wikipedia.org/wiki/Tyrosine

Medical use

Tyrosine is a precursor to neurotransmitters and increases plasma neurotransmitter levels (particularly dopamine and norepinephrine)[8] but has little if any effect on mood.[9][10][11] The effect on mood is more noticeable in humans subjected to stressful conditions (see below).

A number of studies have found tyrosine to be useful during conditions of stress, cold, fatigue,[12] loss of a loved one such as in death or divorce, prolonged work and sleep deprivation,[13][14] with reductions in stress hormone levels,[15] reductions in stress-induced weight loss seen in animal trials,[12] improvements in cognitive and physical performance[10][16][17] seen in human trials. Because tyrosine hydroxylase is the rate limiting enzyme, however, effects are less significant than those of l-dopa.

http://en.wikipedia.org/wiki/Moors

Population genetics

See also: African admixture in Europe and Berbers#Genetic evidence

Shomarka Keita, a biological anthropologist from Howard University, has claimed that populations in Carthage circa 200 BC and northern Algeria 1500 BC were very diverse. As a group, they plotted closest to the populations of Northern Egypt and intermediate to Northern Europeans and tropical Africans. Keita claimed that "the data supported the comments from ancient authors observed by classicists: everything from fair-skinned blonds to peoples who were dark-skinned 'Ethiopian' or part Ethiopian in appearance."[26] Modern evidence showed a similar diversity among present North Africans. Moreover, this "diversity" of phenotypes and peoples was probably due to in situ differentiation, not foreign influxes.

Of course foreign influxes certainly had an impact but they did not replace the indigenous Berber population.[27]

The Y chromosome p49a,f TaqI Haplotype V, which corresponds to Y haplogroup E1b1b1b (M81) —formerly E3b1b, E3b2 and colloquially referred to as the "Berber marker"— has been found among 68.9% of modern Berbers in North Africa and as high as 80% in one group. It is believed to be about 5,600 years old,[28] and to have arrived with the Neolithic expansion from the Near East. M81 is not found in Sub-Saharan Africa. This haplotype has also been observed in as high as 40% of one small group of Andalusians tested. Generally it appears at much lower frequencies among Iberian populations, and lower as distance from North Africa increases.[29]

Y DNA haplogroup E1b1b (formerly E3b) predominates among North African populations; its E1b1b1b subgroup (M81) is identified especially with Berbers. The Vb subtype of p49a,f Haplotype V, apparently corresponding to E3b1b, has been found to occur in two-thirds of the

Haplotype V Southern Iberians, that is, in about a quarter of all Andalusians tested. The frequency of Vb is at its highest among Berbers, and was found to decline rapidly from West to East among North Africans sampled. It is uncommon in France and Italy.[29]

A 2006 mitochondrial DNA study of 12th to 13th century Islamic remains from Priego de Cordoba, Spain, indicates a higher proportion (4%) of sub-Saharan African lineages. This is attributed only partially to the period of Moorish occupation; researchers believe that more ancient migrations from Africa to Europe were more significant.[30]

“ Mitochondrial DNA sequences and restriction fragment polymorphisms were retrieved from three Islamic 12th to 13th century samples of 71 bones and teeth (with >85% efficiency) from Madinat Baguh (today called Priego de Cordoba, Spain).

Compared with 108 saliva samples from the present population of the same area, the medieval samples show a higher proportion of sub-Saharan African lineages that can only partially be attributed to the historic Muslim occupation.

Infact, the unique sharing of transition 16175, in L1b lineages, with Europeans, instead of Africans, suggests a more ancient arrival to Europe from Africa. The present-day Priego sample is more similar to the current south Iberian population than to the medieval sample from the same area. The increased gene flow in modern times could be the main cause of this difference. ”

26. ^ G. Mokhtar."General History of Africa: Ancient Civilizations of Africa", pg 427

27. ^ "Studies of ancient crania from northern Africa", AMERICAN JOURNAL OF PHYSICAL ANTHROPOLOGY 83:35-48 (1990)

28. ^ Arredi et al. (2004), A Predominantly Neolithic Origin for Y-Chromosomal DNA Variation in North Africa, Am J Hum Genet. 2004 August;

75(2): 338–345.

29. ^ a b Nathalie GĂ©rard et al., "North African Berber and Arab Influences in the Western Mediterranean Revealed by Y-Chromosome DNA

Haplotypes", Human Biology, Volume 78, Number 3, June 2006, pp. 307–316.


http://en.wikipedia.org/wiki/Amelanism

Melanins and melanin production

Melanin is a compound found in plants, animals, and protists, and is derived from the amino acid tyrosine. Melanin is a photoprotectant, absorbing the DNA-damaging ultraviolet radiation of the sun. Vertebrates have melanin in their skin and hair, feathers, or scales.

They also have two layers of pigmented tissue in the eye: the stroma, at the front of the iris, and the iris pigment epithelium, a thin but critical layer of pigmented cells at the back of the iris. Melanin is also present in the inner ear, and is important for the early development of the auditory system.[1] Melanin is also found in parts of the brain and adrenal gland.

Aeumelanism

Melanocytes depend on the hormone Melanocortin 1 receptor (MC1R) to signal the production of eumelanin. Loss of melanocortin 1
receptor function or high activity of the MC1R-antagonist, Agouti signalling peptide, can cause the widespread absence of eumelanin. Loss of MC1R function, a recessive trait, has been observed in many species.

In humans, various mutations of the MC1r gene result in red hair, blond hair, fair skin, and susceptibility to sundamaged skin and melanoma.[5] Aeumelanic hair coats, associated with mutations of the MC1R gene, have also been identified in mice,[6] cattle,[7] dogs,[8] and horses.[9] These coat colors are called "yellow" in mice and dogs, "red" in cattle and chestnut in horses. The loss of eumelanin in the coat is, in these species, harmless. The distinction between aeumelanism and hyperphaeomelanism - over abundance of phaeomelanin - is semantic.

Aphaeomelanism

Aphaeomelanism is the abnormal absence of phaeomelanin from the integumentary system and/or eyes.[10] Phaeomelanin is produced by melanocytes in the absence of melanocortin 1 receptor. This absence is mediated by agouti signalling protein, which antagonizes melanocortin 1 receptor. Loss of function of agouti signalling protein can permit unmediated phaeomelanin production, producing a uniformly black-to-brown coat color. This condition can be observed in dogs,[11] cats,[12] and horses.[13]

The appearance of mammals with recessive agouti mutations is typically dense black. As with aeumelanism, the difference between lack of phaeomelanin and abundance of eumelanin is one of words. Some agouti alleles in mice are associated with health defects, but this is not the case in dogs, cats, or horses.

http://en.wikipedia.org/wiki/Pituitary_gland

The pituitary gland, or hypophysis, is an endocrine gland about the size of a pea and weighing 0.5 g (0.02 oz.). It is a protrusion off the bottom of the hypothalamus at the base of the brain, and rests in a small, bony cavity (sella turcica) covered by a dural fold (diaphragma sellae). The pituitary fossa, in which the pituitary gland sits, is situated in the sphenoid bone in the middle cranial fossa at the base of the brain. It is considered a master gland. The pituitary gland secretes hormones regulating homeostasis, including tropic hormones that stimulate other endocrine glands. It is functionally connected to the hypothalamus by the median eminence.

Functions

The pituitary hormones help control some of the following body processes:


http://en.wikipedia.org/wiki/Albino

Albinism (from Latin albus, "white"; see extended etymology, also called achromia, achromasia, or achromatosis) is a congenital disorder characterized by the complete or partial absence of pigment in the skin, hair and eyes due to absence or defect of an enzyme involved in the production of melanin.

Causes and types of albinism
Most forms of albinism are the result of the biological inheritance of genetically recessive alleles (genes) passed from both parents of an individual, though some rare forms are inherited from only one parent. There are other genetic mutations which are proven to be associated with albinism. All alterations, however, lead to changes in melanin production in the body.[1][2]

The chance of offspring with albinism resulting from the pairing of an organism with albinism and one without albinism is low. However, because organisms can be carriers of genes for albinism without exhibiting any traits, albinistic offspring can be produced by two non-albinistic parents. Albinism usually occurs with equal frequency in both genders.[1]

An exception to this is ocular albinism, which it is passed on to offspring through X-linked inheritance. Thus, ocular albinism occurs more frequently in males as they do not have a second X chromosome.[3]

There are two of different forms of albinism; a partial lack of the melanin is known as hypomelanism, or hypomelanosis and the total absence of melanin is known as amelanism or amelanosis.

http://en.wikipedia.org/wiki/Melanism

Melanism and the immune system

Genetic research has shown that melanistic wolves owe their colouration to a mutation that first arose in domestic dogs

Melanism has been found to be linked to beneficial changes in the immune system. The Smithsonian Answer Book: Cats notes that genes for melanism in felines may provide resistance from viral infections and that a viral epidemic may explain the prevalence
of black leopards in Java and Malaysia, and the relatively high incidence of black leopards and black servals in the Aberdares region of Africa.[citation needed] Previously, black furred felines in the Aberdares had been considered a high altitude adaptation due to absorbing more heat.[citation needed]


Hope this fills some of the gaps for you to understand some of the missing functions that you need to be aware of. how the Circadian Rythym affects us.

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